A. History
1. Pain
a. Types. Abdominal pain is an invariable feature of an acute abdomen and presents as one or a combination of three types.
i. Visceral pain develops from stretching or distending of an abdominal viscus or from inflammation. The pain is diffuse and poorly localized. It generally has a gnawing, burning, or cramping quality.
ii. Somatic pain arises from the abdominal wall, the parietal peritoneum, the root of the mesentery, or the diaphragm. It is more intense and better localized than visceral pain.
iii. Referred pain is felt at a site distant from the source of the pain but shares the same dermatome or neurosegment. Referred pain is usually sharp and well localized; thus, it resembles somatic pain.
b. The onset of pain may be instantaneous, or the pain may develop over minutes or even hours. Sudden severe pain characterizes such events as a perforated ulcer, a ruptured viscus, a ruptured ectopic pregnancy, a spontaneous pneumothorax, or a dissecting aortic aneurysm. On the other hand, more gradual development of pain is typical of acute pancreatitis, acute cholecystitis, intestinal obstruction, bowel perforation, diverticulitis, and intraabdominal abscess.
2. Vomiting
usually accompanies an acute abdomen to a variable degree. A clinical rule is that pain precedes vomiting in disorders requiring surgical treatment, whereas vomiting precedes pain in medically treated disorders. Vomiting can be persistent, as with intestinal obstruction. Long-standing obstruction may result in feculent vomiting due to the proximal growth of colonic bacterial flora. Vomiting of bloody material suggests that the bleeding lesion is above the ligament of Treitz.