Hiatal hernia classification
Type I hernias are the most common and account for up to 95% of the total prevalence. Type II, III, and IV hernias are together termed paraesophageal hernias (PEH) and combined account for the remaining 5% of hiatal hernias.
Clinical Findings
As mentioned above, many patients remain asymptomatic, and their hernias are diagnosed accidentally. Large PEH, on the other hand, may cause a wide variety of symptoms such as epigastric discomfort, chest pain, postprandial bloating, dysphagia, or respiratory problems (asthma, cough, or dyspnea caused by chronic aspiration). In addition, patients may experience symptoms due to gastroesophageal reflux (heartburn or regurgitation). Anemia secondary to gastric erosions can also be present.
Diagnosis
Several tests are needed preoperatively to determine the anatomy and physiology of the esophagus and stomach.
Barium Swallow
Upper Endoscopy
An upper endoscopy is also useful to determine if gastric or esophageal inflammation is present and to rule out cancer. Cameron ulcers are ulcerations of the mucosal folds lining the stomach due to extrinsic compression of the diaphragm on the distal neck of a hiatal hernia. Though typically asymptomatic, they may present as acute and severe upper gastrointestinal bleeding.
Abdominal and Chest CT Scan
Esophageal Manometry
Abnormal esophageal motility is common in these patients. A partial fundoplication is preferred if there is severely impaired peristalsis. If the manometry is technically unfeasible or the patient cannot tolerate the catheter, a partial fundoplication should also be performed.
Although patients with PEH usually have pathologic reflux, performing a pH monitoring study does not add relevant information preoperatively. The operation will alter the physiology of the EGJ, and a fundoplication to prevent reflux will be performed regardless of the results of the study.
Cardiac- and pulmonary-related tests are performed on a case-by-case basis, particularly because these patients are often elderly .
Surgical Repair of PEH
Historically, surgical repair has been advocated in all patients with PEH, even when asymptomatic, due to the considerable mortality associated with acute hernia incarceration and strangulation. Currently, nonsurgical management is considered a better alternative in asymptomatic or minimally symptomatic patients because the risk of strangulation is lower than the risk of morbidity associated with the operation. Therefore, surgical repair is indicated mainly for symptomatic PEH [2].
Traditionally, PEH repair required either a laparotomy or thoracotomy, and these approaches were associated with high morbidity. Since its introduction in 1992, the laparoscopic approach has been increasingly embraced due to its improved postoperative outcomes [3, 4]. Nowadays, the vast majority of patients with PEH are managed with a laparoscopic approach.
Laparoscopic PEH Repair
Positioning of the Patient
After induction of general endotracheal anesthesia, an orogastric tube is inserted to keep the stomach decompressed. The patient is positioned supine in low lithotomy position with the lower extremities extended on stirrups, with knees flexed 20–30°. To avoid sliding due to the steep reverse Trendelenburg position used during the entire procedure, a bean bag is inflated to create a “saddle” under the perineum. Pneumatic compression stockings and subcutaneous heparin are always used as prophylaxis against deep vein thrombosis (particularly important as the increased abdominal pressure secondary to the pneumoperitoneum and the steep Trendelenburg position decrease venous return). The surgeon stands between the patient’s legs and the first and second assistants on the left and right side of the operating table, respectively.