Complications, risks, and consequences
Estimated frequency
Most significant/serious complications
Infectiona
Subcutaneous/wound
1–5 %
Intra-abdominal (including subphrenic abscess)
0.1–1 %
Intrathoracic (pneumonia, pleural, mediastinitis)
0.1–1 %
Systemic
0.1–1 %
Late – postsplenectomy sepsis (vaccination)
<0.1 %
Bleeding and hematoma formationa
1–5 %
Conversion to open operation
1–5 %
Diarrhea
1–5 %
Symmetrical pouch dilatation
Adults
1–5 %
Adolescents
5–20 %
Band slippage
1–5 %
Port complications (leakage, migration, tube kink)
1–5 %
Bolus obstruction (serious, requiring removal)
1–5 %
Dysphagia or pseudo-achalasia
1–5 %
Reflux esophagitis/pharyngitis/pneumonitis
1–5 %
Failure of suture/staple line/band
1–5 %
Failure to control excessive weight
1–5 %
Delayed gastric (distal) emptying
1–5 %
Inability to vomit or belch
1–5 %
Gas bloat syndrome
1–5 %
Repeated vomiting
1–5 %
Rare significant/serious problems
Pneumothorax
0.1–1 %
Myocardial ischemia/infarction
0.1–1 %
Gas embolus
0.1–1 %
Diaphragmatic injury/hernia
0.1–1 %
Ulceration stomal/esophageal/gastric/duodenal (early or late)
0.1–1 %
Gastric/esophageal/bowel injury or ischemia (devascularization)/perforation
0.1–1 %
Gastric erosion
0.1–1 %
Pancreatic/liver injury
0.1–1 %
Gastro-cutaneous fistula
0.1–1 %
Small bowel obstruction (early or late)a
0.1–1 %
[Adhesion formation]
Deep venous thrombosis and pulmonary embolism
0.1–1 %
Splenic injurya
0.1–1 %
Conservation (consequent limitation to activity, late rupture)
Splenectomy
Extrusion of band +/− ulceration
0.1–1 %
Nutritional deficiency – anemia, B12 malabsorption
0.1–1 %
Multisystem failure (renal, pulmonary, cardiac failure)
0.1–1 %
Deatha
0.1–1 %
Less serious complications
Pain/tenderness [rib pain (sternal retractor), wound pain]
Acute (<4 weeks)
>80 %
Chronic (>12 weeks)
1–5 %
Paralytic ileus
0.1–1 %
Abdominal distention (acute or chronic)/excessive flatus
1–5 %
Intolerance of large meals (necessity for small meals)a
>80 %
Surgical emphysema
1–5 %
Seroma formation
0.1–1 %
Wound scarring (poor cosmesis/wound deformity)
0.1–1 %
Port site herniae
0.1–1 %
Perspective
See Table 5.1. Laparoscopic adjustable gastric banding is an elective procedure almost exclusively used for treating morbid obesity. The procedure is often technically straightforward, if somewhat challenging, and the complication rate is often determined by the degree of obesity and coexistence of other underlying risk factors such as smoking, diabetes, pulmonary disease, and cardiovascular disease. The success in reducing obesity is well established, with the majority of patients maintaining >60 % excess weight loss (EWL) for 5 years or longer. Both the success and the complication rates are closely related to patient selection. Major problems include wound infection of port sites or the implanted reservoir, atelectasis and pneumonia, injury to other abdominal organs, conversion to open operation, venous thromboembolism, band erosion and slippage, stomal ulceration and bleeding, bolus obstruction or stomal stenosis, dysphagia, and inability to eat.
Major Complications/Consequences
The major complications occurring after laparoscopic gastric banding are symmetrical pouch dilatation (more commonly in adolescents than adults), band slippage, and port complications (leakage, migration, tube kinking). Gastric erosion from the band is rare. Wound infection of port sites or implanted reservoir, chest infection, or intra-abdominal infection are relatively rare but can be serious. Gastric or esophageal perforation and leakage is very unusual and is usually diagnosed on routine contrast radiography on the first postoperative day. Systemic infection and multisystem organ failure may then ensue and is the major cause of mortality when it occurs. Possible conversion to open operation is important to warn the patient about. Bleeding is rarely severe and usually controlled at surgery. Failure of “stomal” function can occur. Occasionally mechanical obstruction needs to be excluded from edema, malpositioning, kinking, or insufficient stoma opening size due to cuff overinflation or symmetrical pouch dilatation patient overeating. Usually, improved function of the proximal pouch occurs after a period of weeks following band deflation and can be shown on subsequent contrast swallow. If it persists, however, laparoscopic repositioning or replacement of the band into a more proximal position is required. Bolus obstruction is not uncommon, with or without stomal stenosis, but usually responds to band deflation, but rarely may require endoscopy for removal of the bolus material. Stomal ulceration may occasionally cause bleeding. Erosion of the band material through the stomach wall is reported but is rarely serious, and the eroded band can be removed endoscopically, if the buckle lies intragastrically, or otherwise dealt with laparoscopically. Reservoir displacement or malfunction, tube kinkage, or leakage from the band or port may require revisional surgery. Splenectomy may be necessary from injury in <1 % of cases. Gas embolus or major vascular or bowel injury are additional serious, although very rare, complications of the laparoscopic approach. Venous thromboembolism is a serious and potentially lethal complication, which is related to obesity and surgery, but appears to be no more common in patients having laparoscopic band surgery than any other form of laparoscopic surgery.
Consent and Risk Reduction
Main Points to Explain
Infection (including peritonitis)
Bleeding
Respiratory infection
Laparoscopic complications
Conversion to open surgery
Long-term banding problems
Band may require removal
Further surgery
Risks without surgery
Open Gastric Bypass
Description
General anesthesia is used. Open gastric bypass surgery is principally for elective reduction of weight in the morbidly obese patient. A midline incision is usually used. The aim of open gastric bypass surgery is to provide a smaller gastric pouch for food in the stomach, promoting early satiety, and to provide a bypass for food from the stomach directly to the more distal small bowel. The absorptive capacity for nutrients (predominantly fat malabsorption) causes steatorrhea which, in addition to weight loss, further discourages intake of fatty foods. The procedure utilizes a linear stapler to create a small proximal gastric pouch (or more recently a divided gastroplasty), to which a Roux-en-Y reconstruction is anastomosed, bypassing the distal stomach, duodenum, and upper small bowel, leaving biliary and pancreatic drainage unchanged. Open gastric bypass is gradually being replaced by laparoscopic methods (see below) in many centers. Patient selection and support is especially important for the success of obesity surgery. The abdominal wall is mass-closed, the subcutaneous tissues closed with absorbable interrupted sutures and the skin closed using continuous subcuticular sutures. Wound drain tubes are used according to surgical preference.
Anatomical Points
The anatomy is essentially fairly constant; however, the obese physique both inside and outside of the abdominal muscle wall can make accessibility problematic. Occasionally, the presence of a shorter mesentery or adhesions may make it difficult for the Roux limb to reach the proximal gastric pouch. When the left lobe of the liver is large and bulky, it can obscure vision. Previous surgery can make further surgery difficult from adhesions or altered anatomy. Redoing open gastric bypass surgery can be challenging. The colon, small bowel, and omentum may overlie the stomach and make access more difficult. Splenic adhesions may restrict mobilization of the stomach and increase risk of splenic injury. Although these organs are at risk, generally these can be readily displaced to enable the procedure to be performed safely. The most dangerous stage of the procedure is creation of the posterior gastric window, as damage and leakage to the proximal stomach, cardia, or esophagus can be difficult to repair and possibly only dealt with by drainage.
Table 5.2
Open gastric bypass complications, risks, and consequences
Complications, risks, and consequences | Estimated frequency |
---|---|
Most significant/serious complications | |
Infectiona | |
Subcutaneous/wound | 5–20 % |
Intra-abdominal (including subphrenic abscess) | 1–5 % |
Intrathoracic (pneumonia, pleural, mediastinitis) | 5–20 % |
Systemic | 0.1–1 % |
Late – postsplenectomy sepsis (vaccination) | <0.1 % |
Bleeding and hematoma formationa | 1–5 % |
Diarrhea | 20–50 % |
Bolus obstruction (serious, requiring removal) | 1–5 % |
Dysphagia or pseudo-achalasia | 1–5 % |
Reflux esophagitis/pharyngitis/pneumonitis | 1–5 % |
Failure of suture/staple line/small bowel anastomotic leakage | 1–5 % |
Failure to control excessive weight | 1–5 % |
Delayed neo-gastric emptying | 20–50 % |
Inability to vomit or belch | 5–20 % |
Gas bloat syndrome | 1–5 % |
Repeated vomiting | 1–5 % |
Nutritional deficiency – anemia, B12 malabsorption | 1–5 % |
Dumping syndrome | 1–5 % |
Early dumping (vasomotor) | |
Late dumping (osmotic, insulin surge) | |
Stomal stenosis | 5–20 % |
Stomal dilatation (widening) | 5–20 % |
Pouch gastritis | 1–5 % |
Pouch dilatation | 1–5 % |
Rare significant/serious problems | |
Stomal ulceration | 0.1–1 % |
Pneumothorax | 0.1–1 % |
Myocardial ischemia/infarction | 0.1–1 % |
Diaphragmatic injury/hernia | 0.1–1 % |
Gastric/esophageal/bowel injury or ischemia (devascularization)/perforation | 0.1–1 % |
Pancreatic/liver injury | 0.1–1 % |
Gastro-cutaneous fistula | 0.1–1 % |
Small bowel obstruction (early or late)a | 0.1–1 %
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